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Summary Glucose-6-phosphate dehydrogenase (G6PD) deficiency leads to an impaired regeneration of reduced glutathione, an important antioxidant, which makes RBCs more susceptible to oxidative stress and can result in episodic hemolytic anemia

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This phenomenon was further intensified in the DBG + US group, revealing the additional enhancement of the function of effector immune cells by N-glycosylation inhibition