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FASEB J 2004;18:11921199

The finding that GSH can enter the ER by facilitated diffusion through the Sec61 translocon in a yeast mutant [36] is congruent with this model, even though it should be acknowledged that the toxic ~ 10-fold increase in cytosolic GSH in this yeast mutant represents a rather non-physiological situation with regard to glutathione gradients at intracellular membranes

For complex multifactorial diseases such as autoimmune disorders, aOoC systems could facilitate the discovery of personalized biomarkers and therapeutic targets by recreating each patients unique genetic, epigenetic, and microenvironmental landscape