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163 At the lesion core therefore, all neural cells die by necrosis, which leads to a massive release of damage-associated molecular patterns (DAMPs), including release of glutamate and ATP, which propagate excitotoxicity

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TRPM7 is a divalent cation channel permeable to Ca 2+ , Mg 2+ , Mn 2+ , and, notably Zn 2+ .[20] Additionally, while carvacrol and 2-APB (TRPM7 inhibitors), reversed neuronal death by reducing Zn 2+ accumulation [21]