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In acquired B12 deficiency, both oral and IM routes have been featured in guidelines to replenish this compound in deficient states

HMNDYT2 SLC39A14 Deficiency In 2016, bi-allelic mutations in SLC39A14 were identified in individuals with features of Mn neurotoxicity, such as rapidly progressing dystonia, variable parkinsonism, and T1 hyperintensity in the globus pallidus observed on brain MRI [45]