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These forms of epigenetic modifications interact with each other to jointly regulate gene expression, playing critical roles in processes such as cell differentiation, development, disease progression, and stress responses [17, 18]

The rebound reflects removal of the temporary antioxidant buffer, not a pathological withdrawal response

In this screen, we finally identified cholesterol and desmosterol (hereafter referred as CH&Desmo) as endogenous ferroptosis inhibitors

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