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~35% inward-open) in the apo state indicates a dynamic equilibrium with a preference for the inward-occluded conformation

Senomorphics: Modulating the Senescence-Associated Secretory Phenotype (SASP) Unlike senolytic therapies that aim to directly eliminate senescent cells, senomorphics work by modulating the senescence-associated secretory phenotype (SASP), which consists of pro-inflammatory cytokines, chemokines, growth factors, and extracellular matrix-degrading enzymes secreted by senescent cells

By introducing electron-donating or electron-withdrawing groups, their pH sensitivity and plasma stability can be finely tunedstable in circulation while rapidly degrading inside target cells