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10.1007/s12192-016-0695-3 209 Zanotto-FilhoA.BraganholE.EdelweissM

Animal seizure models have demonstrated that pharmacological doses of vitamin E administered after a kainate-induced seizure, can almost completely restore function of glutamine synthase, the enzyme responsible for converting excitatory glutamate, into non-toxic glutamine, for transport between astrocytes and neurons ( in vitro data which suggests that vitamin E may be able to reduce activity of the ionotropic AMPA, kainate, and NMDA glutamate receptors, in addition to protecting mitochondrial membrane potential through its antioxidant function ( in vitro and in vivo evidence that vitamin E can reduce neuroinflammatory responses mediated by microglial cells (Li et al., 2001

Target compounds were detected with a 6500 plus QTrap mass spectrometer (AB SCIEX, USA) coupled with a ACQUITY UPLC H-Class system, equipped with a heated electrospray ionization (HESI) probe

In the diet-induced obese mice, the 21 nm cAuNPs demonstrated anti-inflammatory and anti-obesity effects [50]