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Foam cell formation during atherogenesis could be also explained in part by uptake of CRP-opsonized native LDL (193)

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In the present study, we leveraged the well-established molecular and clinical homogeneity of RMC 1,9 , which allowed us to clearly detect an unusually prominent clinical signal of hyperprogression in our prospective clinical trial and subsequently replicate and functionally characterize these findings in our RMC mouse model