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Hip injuries heal slowly because of limited blood supply to deep structures, constant mechanical loading from daily movement, and the tendency for one problem to create compensatory issues in surrounding tissues

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Its primary mechanisms include promoting angiogenesis (new blood vessel formation) through VEGF upregulation, modulating the nitric oxide system in a context-dependent manner, activating the FAK-paxillin pathway to drive cell migration and tissue rebuilding, and regulating multiple growth factors (PDGF, TGF-beta, FGF) involved in organized tissue repair