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We further demonstrated that Dlgap5 overexpression promoted NLRP3/CASPASE1 activation and GSDMD cleavage and pyroptosis, while these effects were abolished by the NF-B inhibitor Bay117082 treatment (Figure 5K), establishing that DLGAP5 initiated NLRP3 inflammasome activation via NF-B pathway

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Bo dziaaj na dwch rnych, lecz dopeniajcych si poziomach organizacji biologicznej w tym samym polu starzeniu i regeneracji tkanki

Cell lines Mouse embryonic fibroblasts (MEFs) and their variants (gift of Dr