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Preclinical studies suggest that GLP-1 signaling in the central nervous system may influence neuronal activity patterns, though the clinical significance of these findings for human fatigue remains theoretical

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Secondly, such deficiency seems to be intimately related to the onset of MetS and establishment of vascular derangements leading to atherosclerosis and finally playing a definitive part in cerebrovascular and myocardial accidents, where IGF1 deficiency seems to render these organs vulnerable to oxidative and apoptotic/necrotic damage

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