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Smoking-induced adipose dysfunction is characterized by altered adipocyte differentiation, impaired insulin action, and dysregulated adipokine secretion (274)

Although the body produces some of its own glutathione, poor diet, smoking, air pollution, stress, infections and radiation reduce the proportion of glutathione in the body

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Fourth, DN involves multiple regulated cell-death pathways with potential crosstalk, and since apoptosis-related proteins (such as the caspase family) and autophagy markers (such as LC3 and p62) were not assessed, it cannot be excluded that DAPA also modulates these pathways and contributes to the observed protective effects