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In the third step, we extracted age group-specific RR data and relevant bias covariates from the studies identified in our systematic review approach of age-specific smoking risk on CVD outcomes, and used MR-BRT to model the age pattern of excess risk (that is, RR-1) of smoking on CVD outcomes with age group-specific excess RR data for all CVD outcomes

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Overall, 25 genes (Table 1) were deregulated by exposure to the three types of emissions, including genes involved in cancer development (increase in Pla1a expression and down-regulation of the tumour suppressor genes Cdkn2a and Ppp2r2c) and cell metabolism (e.g