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This age-dependent variability in oxytocin signaling may delineate a developmental window in which such therapy could be most effective

With aging, the gradual accumulation of atherosclerotic risk factors, including oxidative stress, inflammation, dyslipidemia, and hyperglycemia, drives metabolic reprogramming in ECs, particularly in regions exposed to disturbed flow with OS, ultimately leading to EC dysfunction and atherosclerosis

This finding suggests that the peptides tolerability is not dependent on metabolic dysfunction and may apply across a broader population spectrum

(172) demonstrated in diabetic rat models that TMAO treatment exacerbated renal inflammation and fibrosis through activation of the NLRP3 inflammasome, promoting IL-1 and IL-18 release and increasing pro-fibrotic TGF-1 expression